2016-05-13 17:28:52 UTC

Where Does Metaplasia in the Esophagus and Stomach Originate?

May 17, 2016

Join us for the 2016 Freston Conference to delve deeper into the topic of intestinal metaplasia in the esophagus and stomach.

Over the last few weeks, we’ve previewed sessions one and two of the 2016 James W. Freston Conference, which will take place Aug. 19 through 21 in Chicago, IL. Led by expert faculty, the 2016 conference will delve deeper into the pathogenesis, biology and translational features of intestinal metaplasia in the esophagus and stomach.

This week conference organizers are happy to provide you with an overview of session three. Details are below.

Potential Origins of Metaplasia in the Esophagus and Stomach
Session three aims to provide clarity on the controversial subject of the origin of metaplastic cells. During this session, you'll hear from renowned researchers: 

  • David H. Wang, MD, PhD, who will analyze key anatomical and histological data from rodent models, which can be beneficial in understanding intestinal metaplasia in humans.
  • Ramesh A. Shivdasani, MD, PhD, who will report on how chromatin states found in human cells relate to developmental underpinnings of Barrett's esophagus found in chromatin states of the mouse fetal gut.
  • Andrea Todisco, MD, who will explore the importance of bone morphogenetic proteins in regulating GI epithelial cell growth and differentiation.
  • James R. Goldenring, MD, PhD, AGAF, who will review the evolution of spasmolytic polypeptide-expressing metaplasia and its correlation to intestinal metaplasia in the stomach.
  • Jianwen Que, MD, PhD, who will examine signaling molecules responsible for controlling the development of the esophagus and stomach. Dr. Que will also examine new findings on epithelial morphogenesis in the esophagus.

Stay tuned to gastro.org for details on session four. To register and view the program in its entirety, visit the conference Web page

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